https://nova.newcastle.edu.au/vital/access/ /manager/Index en-au 5 https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:32995 Wed 23 Feb 2022 16:04:08 AEDT ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:4326 Sat 24 Mar 2018 07:18:23 AEDT ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:4327 Sat 24 Mar 2018 07:18:23 AEDT ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:47762 P < .001); the straight sinuses were not significantly different. In idiopathic intracranial hypertension, both sinuses were not significantly different from those of controls. The pressure drop from the sagittal sinus to the end of the straight sinus was elevated by 1.2 mm Hg in hydrocephalus (P = .001) but not significantly different from that in controls in idiopathic intracranial hypertension. Shunt insertion dilated the sagittal sinuses in hydrocephalus, leaving them 18% larger than normal and eliminating the transvenous pressure change. Conclusions: There is a transvenous pressure difference in hydrocephalus that is absent in idiopathic intracranial hypertension. This difference is eliminated by shunt insertion. The findings may have a bearing on ventricular dilation.]]> Fri 27 Jan 2023 10:53:30 AEDT ]]>